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Tissue Doppler Imaging
Strain Rate (SR) Imaging
Non-viable Apical Segments

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Sadeghian, H., Vosoughi, P., Sadeghian, A., Lotfi-Tokaldany, M., Ashrafi, M. M., & Shahrzad, S. (2018). Evaluation of Longitudinal Tissue Velocity and Deformation Imaging in Akinetic Non-viable Apical Segments of Left Ventricular Myocardium. Internal Medicine and Medical Investigation Journal, 3(4). https://doi.org/10.24200/imminv.v2i4.140

Evaluation of Longitudinal Tissue Velocity and Deformation Imaging in Akinetic Non-viable Apical Segments of Left Ventricular Myocardium

Hakimeh Sadeghian
Associate professor of Cardiology, Shariati Hospital , Tehran University of Medical Science, Tehran, Iran
Parastoo Vosoughi
Tehran Heart Center,Tehran, Iran
Afsaneh Sadeghian
Bahar Hospital, Shahrood University of Medical Science, Shahrood, Iran.
Masoumeh Lotfi-Tokaldany
Tehran Heart Center,Tehran, Iran
Mohammad Moein Ashrafi
Medical student, Yazd Branch of Azad University, Faculty of Medicine, Yazd, Iran
Soraya Shahrzad
Associate professor of Cardiology, Shariati Hospital , Tehran University of Medical Science, Tehran, Iran

Introduction: The use of tissue velocity and strain rate imaging is proposed for the quantification of non-viable myocardium. This study is aimed at investigating the differences in tissue velocity and strain rate imaging indices between non-viable left ventricular apical segments and the normal segments using segment-by-segment comparison.

Materials and Method: Thirty-two patients with akinetic left ventricular apical segments and without viability were selected using two-dimensional echocardiography and dobutamine stress echocardiography; 32 individuals with normal echocardiography and coronary angiography formed the normal group. Peak systolic velocity, peak systolic strain, and strain rate were measured in the four left ventricular apical segments and the apex 17th segment.

Results: The patient group had a significantly lower ejection fraction (26.88±6.06% vs. 56.56±2.36%; p<0.001). Overall, the patient group had significantly lower resting peak systolic velocity, systolic strain, and strain rate. In the segment-by-segment comparison, only systolic strain showed a remarkable reduction in the patient group, while reduction in Sm and strain rate were not significant in all the segments. After dobutamine stress echocardiography, only systolic strain showed an insignificant increase compared to the resting values. In the apex 17th segment, Sm showed significant reduction in the patient group.

Conclusion: The ST in apical segments may be used as a quantitative index for detecting akinesia both at rest and after dobutamine infusion. Reduction in Sm can be used as a marker of akinesia in the apical cap at rest.



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